Health Risk Assessment on Ambient Air Pollution of Particles in Taiwan (II)
「Health Risk Assessment on Ambient Air Pollution of Particles in Taiwan (II)」於資料集「EPQSummary_ChemicalSubstanceManagement」由單位「行政院環境保護署」的陳先生所提供，聯繫電話是02-2311-7722#2386，最近更新時間為：2023-07-30 01:01:48。 欄位Project Title的內容是Health Risk Assessment on Ambient Air Pollution of Particles in Taiwan (II) ， 欄位Project Subject的內容是The project entitled “Health Risk Assessment on Ambient Air Pollution of Particles in Taiwan Part II.” is a continuous project of a three-year research project entitled “Health Risk Assessment on Ambient Air Pollution of Particles in Taiwan.” and “Asian Continental Dust Storm -Induced Health Effects in Taiwan.” In the project of “Health Risk Assessment on Ambient Air Pollution of Particles in Taiwan.” we establish a Supersite and a “Particle toxicological laboratory”. In the project of “Asian Continental Dust Storm-Induced Health Effects in Taiwan.” we establish “Surveillance net of emergency case” and “Surveillance net of susceptible population” to evaluate the relationship between dust storm and subjects. By applying these critical research techniques to particle researches, we have achieved the following four specific research objectives. First, the Supersite monitoring during 1/1/2003-10/31/2002 have provided new and detailed size-specific number and mass PM concentration. Significantly high PM10, PM2.5, total carbon, organic carbon, and nitrate concentrations have been observed during the morning and afternoon commute periods. Second, concentrated ambient particles in Shin-Chuan City of Taipei County didn’t show any significant cardio-circulatory toxicity on diseased rat models. Such results were due to the effects of transportation for disease rats from Laboratory Animal Center to Shin-Chuan City. Third, PM collected from different pollution sources induced different levels of interleukin-6 (IL-6) and tumor necrosis factor-alpha (TNF-α) released from BEAS-2B cells. PM from straw and Chinese incense has greater cellular toxicity than PM from tobacco. Forth, we found decreases in IL-6 t-PA (tissue plasminogen activator) and PAI-1 (plasminogen activator inhibitor-1) among patients with coronary artery disease during the dust storm period in 2003. We also found an increase in fibrinogen and a decrease in log10pNN50 (percent of adjacent NN intervals differing by more than 50ms) among patients with diabetes during the dust storm period. ， 欄位Project Year的內容是2003 ， 欄位Organizer的內容是Department of Air Quality Protection ＆amp; Noise Control ， 欄位Executive Unit的內容是台灣大學公共衛生學院職業醫學與工業衛生研究所 ， 欄位Reporting download URL的內容是https://epq.epa.gov.tw/ProjectDoc/FileDownload?fid=2598&proj_id=A000000321105&doc_kind=1 ， 欄位Publish Date的內容是20040301 。
Project Title
Health Risk Assessment on Ambient Air Pollution of Particles in Taiwan (II)
Project Subject
The project entitled “Health Risk Assessment on Ambient Air Pollution of Particles in Taiwan Part II.” is a continuous project of a three-year research project entitled “Health Risk Assessment on Ambient Air Pollution of Particles in Taiwan.” and “Asian Continental Dust Storm -Induced Health Effects in Taiwan.” In the project of “Health Risk Assessment on Ambient Air Pollution of Particles in Taiwan.” we establish a Supersite and a “Particle toxicological laboratory”. In the project of “Asian Continental Dust Storm-Induced Health Effects in Taiwan.” we establish “Surveillance net of emergency case” and “Surveillance net of susceptible population” to evaluate the relationship between dust storm and subjects. By applying these critical research techniques to particle researches, we have achieved the following four specific research objectives. First, the Supersite monitoring during 1/1/2003-10/31/2002 have provided new and detailed size-specific number and mass PM concentration. Significantly high PM10, PM2.5, total carbon, organic carbon, and nitrate concentrations have been observed during the morning and afternoon commute periods. Second, concentrated ambient particles in Shin-Chuan City of Taipei County didn’t show any significant cardio-circulatory toxicity on diseased rat models. Such results were due to the effects of transportation for disease rats from Laboratory Animal Center to Shin-Chuan City. Third, PM collected from different pollution sources induced different levels of interleukin-6 (IL-6) and tumor necrosis factor-alpha (TNF-α) released from BEAS-2B cells. PM from straw and Chinese incense has greater cellular toxicity than PM from tobacco. Forth, we found decreases in IL-6 t-PA (tissue plasminogen activator) and PAI-1 (plasminogen activator inhibitor-1) among patients with coronary artery disease during the dust storm period in 2003. We also found an increase in fibrinogen and a decrease in log10pNN50 (percent of adjacent NN intervals differing by more than 50ms) among patients with diabetes during the dust storm period.
Project Year
2003
Organizer
Department of Air Quality Protection ＆amp; Noise Control
Executive Unit
台灣大學公共衛生學院職業醫學與工業衛生研究所
Reporting download URL
https://epq.epa.gov.tw/ProjectDoc/FileDownload?fid=2598&proj_id=A000000321105&doc_kind=1
Publish Date
20040301
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